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Empyrean Vector V2 - Alpha-Lipoic Acid and Much More

By now there is a battery of research available on alpha-lipoic acid, and one form in particular shows the most promise: Na-R-ALA — sodium-stabilized, R-enantiomer alpha-lipoic acid.

Chemistry, anyone?

Chemistry, anyone?

Quickly, the R-enantiomer (+) is the one that is used by the body, but if you were to buy basic alpha-lipoic acid (ALA) you would be getting what is known as the racemic mixture of ALA. Racemic simply means a 50/50 mixture of the R-enatiomer (+) and the S-enantiomer (-). To avoid getting too technical, what’s important here is that the S-enantiomer (-) is not found in nature and may inhibit the R-enantiomer (+) from interacting with proteins, enzymes, and genes, and thus is undesirable. Of primary significance, R-enantiomer alpha-lipoic acid has shown to be ten times more effective than racemic alpha-lipoic acid for reducing inflammation. Furthermore, adding the sodium group (Na+) makes the compound water-soluble and further enhances absorption, as well as extending the shelf-life to up to three years without refrigeration.

Here are some of the purported benefits of Na-R-ALA:

  • Protect both the fatty and aqueous portions of cells from harmful free radicals
  • Reverse oxidative damage to enzymes and DNA
  • Restore levels of glutathione, a protective antioxidant and detoxification compound
  • Increase ATP (energy) levels
  • Support a healthy response to inflammation, including decreasing C-reactive protein
  • Enhance cardiovascular health by promoting vasodilation (relaxation of blood vessels)
  • Improve metabolism, increase insulin sensitivity and improve glucose transport
  • Reduce appetite, decrease fat accumulation and increase lean muscle mass
  • Protect the integrity of the brain and nervous system
  • Chelate heavy metals

In this form, this is definitely a supplement worth taking a second look at, if not a staple. There is even some interesting, although not yet conclusive, research showing that Na-R-ALA can induce apoptosis (cell death) in human colon cancer cells.

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